Disc edema, transient obscurations of vision, and a temporal fossa mass

Katz, Barrett

doi:10.1016/0039-6257(91)90126-z

Cited by 15 publications

(6 citation statements)

References 50 publications

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“…Intracranial, noncaseating granulomata of sufficient size or at significant locations may mimic intracranial tumors on neuroimaging 3 and can raise ICP, causing papilledema. 4 Granulomatous meningoencephalitis can elevate ICP sufficiently to cause papilledema and may have an acute, subacute, or chronic course. Patients with acute sarcoid meningoencephalitis may develop seizures and changes in mental status, ranging from confusion to coma and death.…”

Section: Papilledemamentioning

confidence: 99%

The Pathogenesis and Treatment of Optic Disc Swelling in Neurosarcoidosis

Katz

Bruno²,

Winterkorn³

et al. 2003

Arch Neurol

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Section: Papilledemamentioning

confidence: 99%

The Pathogenesis and Treatment of Optic Disc Swelling in Neurosarcoidosis

Katz

Bruno²,

Winterkorn³

et al. 2003

Arch Neurol

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“…In other cases of sarcoidosis presenting with basal space-occupying masses, meningioma has been suspected preoperatively. [22][23][24] Katz 22 described a 56year-old woman with a mass in the middle cranial fossa that was enhanced after the administration of gadolinium. Lexa and Grossman 23 reviewed MRI scans in 24 patients with the diagnosis of sarcoidosis and symptoms referable to the brain and found that 2 patients had extraaxial masses mimicking meningioma.…”

Section: Pathological Discussionmentioning

confidence: 99%

Case 37-1996

Gittinger¹,

Hedley‐Whyte²

1996

N Engl J Med

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A 51-year-old man was admitted to the hospital because of visual "instability" and an intracranial mass.The patient was an accountant. He had been well until six years earlier, when he began to have difficulty fixating on the proper line when reading pages with columns of figures. The problem was bilateral but appeared to be worse in the left eye. Five years before admission, he noticed a progressive loss of vision in the left eye. Two years later, he experienced anosmia and had urinary frequency and urgency, with nocturia five times nightly. Several days before admission to this hospital, the patient entered another hospital, where a cranial magnetic resonance imaging (MRI) scan ( Fig. 1 and 2) showed a densely enhancing extraaxial subfrontal mass, 5.5 cm by 2.2 cm by 1.7 cm on T 2 -weighted images. The mass, which was isointense on T 1 -weighted images and hypointense on T 2 -weighted images, extended along the falx into the interhemispheric fissure, along the inferior surface of the left basal ganglia and hypothalamus, and into the suprasellar region, where it surrounded the left optic chiasm and abutted the left aspect of the pituitary stalk. The frontal lobes were involved, with extensive vasogenic edema in the white matter of the inferior frontal lobes and in the external capsules. The mass encased and narrowed the distal left internal carotid artery, the proximal portions of both anterior cerebral arteries, and the left middle cerebral artery. Scattered, nonspecific hyperintense areas were seen in the periventricular white matter. There was a retention cyst in the right maxillary sinus, with mucosal thickening in the left maxillary and bilateral ethmoid sinuses. The patient was referred to this hospital.He had worked as a paint stripper 15 years earlier, with prolonged exposure to solvents. There was a six-year history of sinusitis with chronic, clear postnasal drip. He had smoked a pipe and cigars in the past but did not smoke cigarettes; he drank small amounts of alcohol. There was no history of diplopia, headache, dizziness, dysarthria, dysphagia, epistaxis, risk factors for human immunodeficiency virus (HIV) infection, foreign travel, intolerance of heat or cold, changes in sexual function, impaired judgment, or altered personality. There was a history of parkinsonism in his father but no family history of other neuromuscular disease or cancer. The temperature was 37.2°C, the pulse was 68, and the respirations were 18. The blood pressure was 150/90 mm Hg.On physical examination, no rash or lymphadenopathy was found. The head was normal, and the neck supple. The lungs, heart, abdomen, and extremities were normal; no evidence of arthritis was found. The rectal examination was normal except for slight prostatic enlargement without nodules.

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“…33 Approximately 70% of patients with papilledema will experience transient visual obscurations (TVO) that usually occur with postural changes but may arise spontaneously. [34][35][36] TVO typically last seconds to minutes, involve one or both eyes, and resolve completely. The pathophysiology of these episodes remains unknown but may be related to ischemia or distortion of myelin at the nodes of Ranvier resulting in reversible nerve dysfunction.…”

Section: Atrophic Papilledemamentioning

confidence: 99%