Distribution of lectin receptors on the plasma membranes of brain glioma cells and autologous peripheral blood mononuclear cells as a function of the degree of anaplasia

Abstract

1035tumor and native cells and between the number of type I and II epithelial cells in affected and unaffected tissue, These findings point at homeostatic stability of epithelial-mesenchymal structures in breast carcinoma, prompting the search for new approaches to its regulation.The content of the D-mannose-specific LCL-receptor that binds D-mannose-containing lymphokines (interleukin-1 and interleukin-2) is proportional to the degree of glioma anaplasia. This may reflect the mechanism whereby brain glioma utilizes lymphokines to proliferate and to escape immunologic surveillance.