2021
DOI: 10.3390/ijms22020905
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Factors Affecting the Progression of Infection-Related Glomerulonephritis to Chronic Kidney Disease

Abstract: Acute glomerulonephritis (AGN) triggered by infection is still one of the major causes of acute kidney injury. During the previous two decades, there has been a major paradigm shift in the epidemiology of AGN. The incidence of poststreptococcal acute glomerulonephritis (PSAGN), which develops after the cure of group A Streptococcus infection in children has decreased, whereas adult AGN cases have been increasing, and those associated with nonstreptococcal infections, particularly infections by Staphylococcus, … Show more

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Cited by 18 publications
(22 citation statements)
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“…In addition, in most IRGN cohorts underlying diabetes is a risk factor for kidney failure. 3,27,28 Majority (88%) of our cohort had underling diabetic kidney disease with almost half of them having advanced DKD. Similar findings were noted in previous studies.…”
Section: Discussionmentioning
confidence: 99%
“…In addition, in most IRGN cohorts underlying diabetes is a risk factor for kidney failure. 3,27,28 Majority (88%) of our cohort had underling diabetic kidney disease with almost half of them having advanced DKD. Similar findings were noted in previous studies.…”
Section: Discussionmentioning
confidence: 99%
“…Although only small amounts of subepithelial EDD were observed in the renal biopsy tissues of our patients, it is not so rare for the limited renal biopsy tissues of IRGN patients not to detect typical humps, and actually we have previously reported a case of a patient with PSAGN in which renal biopsy showed atypical massive subendothelial deposits without subepithelial humps ( 16 ). The differences in responses of the affected hosts, including genetic and/or acquired background of the complement system, may affect the specific histology ( 17 ), especially in the later phase of disease course.…”
Section: Discussionmentioning
confidence: 99%
“…La asociación ente el hacinamiento y el número de convivientes con la proteinuria, podría deberse a la persistencia de la infección, lo que resulta en la continuación del mecanismo patogénico [21]. Hoy et al [4] reportaron que la presencia de anticuerpos antiestreptocócicos específicos y persistentes, evaluada en un número limitado de sujetos seleccionados, se asoció con niveles más altos de proteinuria [4].…”
Section: Discussionunclassified