Hl-a Antigen 27 and Reactive Arthritis

Aho, Kimmo; Ahvonen, P; Lassus, A; Sievers, K; Tiilikainen, A

doi:10.1016/s0140-6736(73)93109-7

Cited by 211 publications

(53 citation statements)

References 10 publications

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“…DISCUSSION A high proportion of patients with ankylosing spondylitis (14,15) and their relatives (14) possess the histocompatibility antigen HL-A 27, whereas this antigen is relatively rare in control populations. HL-A 27 is also associated highly significantly with the more acute yersinia arthritis and Keiter's disease (11,(16)(17)(18) and anterior uveitis (19). T h e present communication extends our previous observations (11) on HL-A 27 in cases of reactive arthritis after certain verified infections.…”

Section: Resultssupporting

confidence: 86%

HL‐A 27 in reactive arthritis. A study of yersinia arthritis and Reiter's disease

Aho

Ahvonen

Lassus

et al. 1974

Arthritis & Rheumatism

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242

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The histocompatibility antigen HL‐A 27 was identified in 43 of 49 patients with yersinia arthritis and in 36 of 40 patients with Reiter's disease, compared with 3 of 20 patients with yersinia infection without arthritis and 14% of the normal Finnish population. HL‐A 2 occurred in patients with reactive arthritis in the same high frequency as did HL‐A 27, but this antigen is present in 55% of Finns. HL‐A 27 negative patients usually had a mild or somewhat atypical disease.

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Section: Resultssupporting

confidence: 86%

HL‐A 27 in reactive arthritis. A study of yersinia arthritis and Reiter's disease

Aho

Ahvonen

Lassus

et al. 1974

Arthritis & Rheumatism

Self Cite

242

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“…These are the same subgroups who have a high incidence of a seronegative arthritis. These subgroups have this in common with Reiter's disease [1,16], anchylosing spondylitis [6] and Yersinia arthritis [1] indicating a link between these entities. It should be noted that in all these groups sacroileitis is a common finding.…”

Section: Discussionmentioning

confidence: 93%

HL-A Antigens in Pustular Psoriasis

et al. 1977

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HL-A typing was performed on 97 patients with pustular psoriasis. HLA-B27 was found increased for the combined three subgroups: localized psoriasis of palms and soles, acrodermatitis continua and generalized pustular psoriasis, who were associated with a high incidence of arthritis. These subgroups have this in common with Reiter’s disease indicating a link between the entities. In persistent palmo-plantar pustulosis an increased incidence of HLA-Bw35 was found. HLA-B13, HLA-B17 and HLA-Bw37 which are found markedly increased in psoriasis vulgaris were in acrodermatitis continua, generalized pustular psoriasis and persistent palmo-plantar pustulosis either absent or not increased as compared with the control population. 7 of 30 patients with localized psoriasis of palms and soles had one of these antigens. Our findings confirm that psoriasis vulgaris and pustular psoriasis as such, seem to be different aetiological entities. Some patients with localized psoriasis of palms and soles may be true psoriatics which besides their psoriasis have a tendency to develop a pustular reaction in their palms and soles similar to persistent palmo-plantar pustulosis.

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“…Another patient had, within 1 month, first the onset of rheumatoid arthritis and then Yersinia arthritis. The benign course of reactive arthritis has been emphasized by some (13,14), but not all (5,(15)(16)(17), authors. The good initial prognosis of reactive arthritis may be changed if there are recurrent attacks of arthritis.…”

Section: Discussionmentioning

confidence: 99%

Ten‐year followup study of patients with yersinia arthritis

Leirisalo‐Repo

Suoranta

1988

Arthritis & Rheumatism

106

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Eighty‐five patients with acute Yersinia arthritis were seen in followup for a mean of 10 years. During that time, peripheral joint symptoms occurred frequently (51.8%), but these symptoms were mild (45.9%). Development of a new reactive arthritis (4.7%) or chronic arthritis (2.4%) was uncommon. One‐third of the patients experienced low back pain, and one‐third of the patients had radiologic evidence of sacroiliitis. The presence of sacroiliitis was more frequent in patients with low back pain (46.7%) than in those who did not have symptoms (21.2%). More patients with HLA–B27 had low back pain and sacroiliitis, but there was no association of this genetic factor with the residual symptoms in peripheral joints.

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Hl-a Antigen 27 and Reactive Arthritis

Cited by 211 publications

References 10 publications

HL‐A 27 in reactive arthritis. A study of yersinia arthritis and Reiter's disease

HL‐A 27 in reactive arthritis. A study of yersinia arthritis and Reiter's disease

HL-A Antigens in Pustular Psoriasis

Ten‐year followup study of patients with yersinia arthritis

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