Identification of groups at risk for renal diseases (including nephrotoxicity)

Dieperink, Hans

doi:10.1016/0378-4274(89)90134-3

Cited by 6 publications

(2 citation statements)

References 35 publications

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“…In fact, individuals at risk are not only those who are in occupations with the highest burden of nephrotoxin exposure. 12 Lower socioeconomic status appears to be independently associated with increased risk for CKD. 6,7,13 Additionally, high-stress behaviors have been found to be associated with increased sympathetic activity 14 that in turn, has been found to be associated with higher risk for initial stage renal disease as well as advanced stage renal failure.…”

Section: Introductionmentioning

confidence: 99%

“…The reported extent of occupational effects on CKD has been suggested to be underestimated. 12 It was reported that of all occupation-related CKD deaths, about 8.2%-14.5% are attributable to silica, organic solvents, lead, and cadmium exposures. 19 Occupations such as cashier, sales representative, carpenter, and manager have been found to be associated with the increased mortality risk for CKD.…”

Section: Introductionmentioning

confidence: 99%

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Occupational risk and chronic kidney disease: a population-based study in the United States adult population

Rubinstein¹,

Wang²,

2013

IJNRD

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Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Occupational risk and chronic kidney disease: a population-based study in the United States adult population

Rubinstein¹,

Wang²,

2013

IJNRD

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Occupational exposure to lead, kidney function tests, and blood pressure

Santos

Colacciopo

Bó

et al. 1994

American J Industrial Med

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In the present study we examined sensitive biochemical markers of kidney function and damage in 166 workers exposed to lead and in 60 control workers. The objective was to investigate the chronic renal toxicity of lead and its possible correlation with arterial pressure. Diastolic arterial pressure was higher in the exposed group (p < 0.05), but the two groups did not differ in systolic pressure. Median activity of urinary N-acetyl-beta-D-glucosaminidase was higher in the exposed group (p < 0.001), and correlated with blood lead levels (p < 0.001) and duration of exposure (p < 0.001), but not with arterial pressure. The other indicators studied, gamma-glutamyl-transpeptidase and alanine-aminopeptidase activity, urine albumin, and total urine protein, were not higher than in the control group and were not correlated with blood lead, duration of exposure, or arterial pressure.

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