Low dietary iron restores functional capabilities of both exocrine and endocrine pancreas in copper-deficient rats

Fields, Meira; Lewis, Charles G.

doi:10.1002/(sici)1520-670x(1997)10:1<1::aid-jtra1>3.0.co;2-h

Cited by 3 publications

(2 citation statements)

References 32 publications

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“…Evidence points to an impaired pancreatic release of insulin as the cause [162][163][164]. Evidence points to an impaired pancreatic release of insulin as the cause [162][163][164].…”

Section: Glycationmentioning

confidence: 99%

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Cardiovascular effects of dietary copper deficiency

Saari

Schuschke

1999

BioFactors

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Dietary copper deficiency may impair cardiovascular health by contributing to high blood pressure, enhancement of inflammation, anemia, reduced blood clotting and arteriosclerosis. The purpose of this review is to compile information on the numerous changes of the heart, blood and blood vessels that may contribute to these cardiovascular defects. These alterations include weakened structural integrity of the heart and blood vessels, impairment of the use of energy by the heart, reduced ability of the heart to contract, altered ability of blood vessels to control their diameter and to grow, and altered structure and function of circulating blood cells. The fundamental causes of these changes rest largely on reduced effectiveness of enzymes that depend on copper for their activity.

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“…Evidence points to an impaired pancreatic release of insulin as the cause [162][163][164]. Evidence points to an impaired pancreatic release of insulin as the cause [162][163][164].…”

Section: Glycationmentioning

confidence: 99%

“…The altered carbohydrate metabolism that is prerequisite to increased glycation is signaled by a reduced glucose tolerance in copper-deficient animals. Evidence points to an impaired pancreatic release of insulin as the cause [162][163][164].…”

Section: Glycationmentioning

confidence: 99%

Cardiovascular effects of dietary copper deficiency

Saari

Schuschke

1999

BioFactors

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Early and advanced glycation end-products are increased in dietary copper deficiency

Saari

Dahlen

1999

The Journal of Nutritional Biochemistry

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Nutritional Factors Adversely Influencing the Glucose/Insulin System

Fields

1998

Journal of the American College of Nutrition

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For the majority of people, particularly if they do not smoke, the food they eat is the largest controllable factor determining their long-term health. The disproportionate consumption of foods high in fat, especially high in saturated fat, and high in simple sugars at the expense of foods high in complex carbohydrate and unsaturated fat has the potential of inducing abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases. Some of the effects of individual macronutrients such as fat, refined sugars and alcohol in promoting abnormalities in glucose/insulin system are presented. These nutrients were chosen because they also have the ability to alter oxidative state of the individual, which in turn could affect the glucose/insulin system. This review focuses on the role of dietary nutrient interactions in influencing the glucose/insulin system through the generation of reactive oxygen species. The importance of dietary macronutrient interaction with micronutrients such as copper and iron and the potential it has in affecting the glucose/insulin system is addressed.

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Low dietary iron restores functional capabilities of both exocrine and endocrine pancreas in copper-deficient rats

Cited by 3 publications

References 32 publications

Cardiovascular effects of dietary copper deficiency

Cardiovascular effects of dietary copper deficiency

Early and advanced glycation end-products are increased in dietary copper deficiency

Nutritional Factors Adversely Influencing the Glucose/Insulin System

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