Morphologische Beitr�ge zur Pathophysiologie des Lymphgef��systems der Leber

Huth, F; Wilde, Annick De; Schulten, H.-J.; Berger, Susanne

doi:10.1007/bf00549031

Cited by 12 publications

(3 citation statements)

References 35 publications

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“…The same is probably true for lymphatic dilatation. Although regeneration of lymphatics after heterotopic small bowel transplantation has been demonstrated in rats, it is only assumed to occur after OTL in humans [8]. It is thought that drainage is reestablished via perihilar lymphangiogenesis, and via peripheral routes available from pericapsular omental adhesions.…”

Section: Discussionmentioning

confidence: 99%

Dilated periportal lymphatics mimicking an anastomotic bile leak after liver transplantation

Rabin

Abramson

Manzarbeitia

et al. 1991

Gastrointest Radiol

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Section: Discussionmentioning

confidence: 99%

Dilated periportal lymphatics mimicking an anastomotic bile leak after liver transplantation

Rabin

Abramson

Manzarbeitia

et al. 1991

Gastrointest Radiol

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“…Die in Gruppe 6 beschriebenen histologischen Verfinderungen wie Erweiterung der Lymphgef~iBe, lymphostatische Odeme und lymphostatische Hfimangiopathie stimmen gut fiberein mit den bisherigen Ergebnissen bei der experimentellen Lymphgef~iBblockade [5,10,11,15]. )~hnlich wie bei den biochemischen Befunden zeigen sich auch bei den histologischen Befunden in Gruppe 6 (Ischfimie und Lymphostase) deutlich st/irker ausgeprS_gte und l/inger anhaltende Ver~inderungen als in Gruppe 4 (Ischfimie).…”

Section: Diskussionunclassified

Potenzierung des tempor�ren, isch�misch bedingten Leberschadens durch eine Lymphabflu�blockade

Wirth

Lie

1982

Langenbecks Arch Chiv

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In order to determine the influence of complete blockage of lymph flow on temporary ischemic liver damage, we operated on 46 mongrel dogs. In group 1, we induced liver ischemia in 4 dogs for 60 min by occlusion of the afferent hepatic vessels (hepatic artery and portal vein), subsequent to portal decompression by a portocaval H-shunt. In group2, we carried out an additional lymph flow blockage by dissection of all connective tissue from the liver in 4 dogs. As only one dog survived in group 2, further experiments were performed with 45-min ischemia. In groups 3 and 4, we induced 45-min ischemia with subsequent biochemical and histological studies, respectively, for 5 weeks postoperatively in a total of 20 dogs. Groups 5 and 6 were treated like groups 3 and 4, but with additional blockage of lymph flow. In groups 3 and 4, no animals died, whereas 4 of 18 dogs died in groups 5 and 6. We found much higher levels of enzymes (GOT up to 1124.8 + 560.9; GPT up to 1139.9 + 475.8 and alkaline phosphatase up to 896.8 + 264.8mU/ml) in group5 than in group3 (GOT up to 88.7+26.9; GPT up to 94.8+27.4 and alkaline phosphatase up to 60.9 + 10.8 mU/ml). In group 3, values returned to normal 3-5 days after surgery compared to 3 weeks and longer in group 5. Besides necrosis and cell infiltrations the histological studies showed edematous changes and a lymphostatic hemangiopathy. The histological damage corresponded to the biochemical changes. It was concluded that additional blockage of lymph flow leads to potential ischemic liver damage.

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“…Unterbindung des Ductus choledochus, z.T. unter zusfitzlicher VerSdung yon LymphgefN3en, ist yon zahlreichen Autoren histologisch, histochemisch, elektronenoptisch und auch zellkinetisch untersucht worden (MacDonald u. Pechet, 1961;Huth et al, 1970;Jones et al, 1976;Carlson et al, 1977;Ogasawara et al, 1978).…”

Section: Regenerationsvorgdnge An Der Durch Ductus-choledochus-unterbunclassified

Die Wundheilung der Leber nach biliärer Obstruktion und Kryonekrose

Helpap

Söhngen

Breining³

1979

Res. Exp. Med.

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Morphologische Beitr�ge zur Pathophysiologie des Lymphgef��systems der Leber

Cited by 12 publications

References 35 publications

Dilated periportal lymphatics mimicking an anastomotic bile leak after liver transplantation

Dilated periportal lymphatics mimicking an anastomotic bile leak after liver transplantation

Potenzierung des tempor�ren, isch�misch bedingten Leberschadens durch eine Lymphabflu�blockade

Die Wundheilung der Leber nach biliärer Obstruktion und Kryonekrose

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