“…Little is known about neurological EM prevalence, but 20-30% has been reported, some cases preceding IBD and some being diagnosed after their remission [13-15]. Several pathogenic mechanisms have been suggested to explain the affectation of peripheral and central nervous system by IBD: malabsorption, deficiency of vitamins, formations of toxic agents, direct effects of the treatment (infections, complications of the immunosuppression), thromboembolism disease and immunological problems [13,14]. Peripheral neuropathies related to IBD seems to be more frequent in UC, with a reported incidence of 1,9%.…”