Possible Association of Atrophic Gastritis and Arterial Stiffness in Healthy Middle-Aged Japanese

Torisu, Takehiro; Takata, Yutaka; Ansai, Toshihiro; Matsumoto, Takayuki; Sonoki, Kazuo; Soh, Inho; Awano, Shuji; Yoshida, Akihiro; Hamasaki, Tomoko; Kagiyama, Shuntaro; Nakamichi, Ikuo; Ohsumi, Tomoko; Toyoshima, Kuniaki; Nishihara, Tatsuji; Iida, Mitsuo; Takehara, Tadamichi

doi:10.5551/jat.943

Cited by 20 publications

(22 citation statements)

References 43 publications

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“…Although the relationship between H. pylori status and adiponectin has not been reported yet, there is one recent report on the circulating levels of adiponectin in patients with atrophic gastritis, in which the cause in most cases was H. pylori infection . Torisu et al.…”

Section: Discussionmentioning

confidence: 99%

Impact of Helicobacter pylori Eradication on Circulating Adiponectin in Humans

Ando¹,

Ishikawa

Takagi

et al. 2012

Helicobacter

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Section: Discussionmentioning

confidence: 99%

Impact of Helicobacter pylori Eradication on Circulating Adiponectin in Humans

Ando¹,

Ishikawa

Takagi

et al. 2012

Helicobacter

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“…Torisu et al 67) reported that CAVI was significantly higher in atrophic gastritis-positive patients than in atrophic gastritis-negative patients, even after adjusting for possible confounding factors (8.59 1.20 vs. 8.27 1.19 p 0.022).…”

Section: H Miscellaneous Diseases And/or Conditionsmentioning

confidence: 99%

Cardio-Ankle Vascular Index (CAVI) as a Novel Indicator of Arterial Stiffness: Theory, Evidence and Perspectives

Shirai

Hiruta

Song

et al. 2011

J Atheroscler Thromb

404

426

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The cardio-ankle vascular index (CAVI) is a new index of the overall stiffness of the artery from the origin of the aorta to the ankle. The most conspicuous feature of CAVI is its independence of blood pressure at the time of measurement. CAVI increases with age and in many arteriosclerotic diseases, such as coronary artery disease, carotid arteriosclerosis, chronic kidney disease and cerebrovascular disease, and is related to many coronary risk factors, such as hypertension, diabetes mellitus, dyslipidemia and smoking. Furthermore, CAVI decreases by controlling diabetes mellitus and hypertension, and also by abstaining from smoking. This suggests that CAVI is a physiological surrogate marker of athero-or arteriosclerosis, and also might be an indicator of lifestyle modification. Recently, it has been reported that CAVI and several left ventricular functions are co-related, suggesting a connection between the heart muscle and vascular function. This review covers the principles of CAVI and our current knowledge about CAVI, focusing on its roles and future outlook. J Atheroscler Thromb, 2011; 18:924-938.

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“…Pulse wave velocity, carotid IMT and FMD were not different in patients with AIG who were negative and positive for H. pylori infection. Torisu et al compared 210 H. pylori positive patients with 133 H. pylori negative patients in terms of PWV [38]. However, they did not find an association between PWV changes and H. pylori status.…”

Section: Discussionmentioning

confidence: 96%