Psychotropic drugs and bruxism

Falisi, Giovanni; Rastelli, C; Panti, F; Maglione, H; Arcega, Raul Quezada

doi:10.1517/14740338.2014.947262

Cited by 72 publications

(56 citation statements)

References 50 publications

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“…prosthodontist ) who was blind to diagnosis under the same conditions. The second assessor separately examined and measured the 30 cause bruxism, but international literature supports the correlation between medications and bruxism 31 .…”

Section: Methodsmentioning

confidence: 99%

Signs of bruxism and temporomandibular disorders among patients with bipolar disorder

et al. 2017

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Section: Methodsmentioning

confidence: 99%

Signs of bruxism and temporomandibular disorders among patients with bipolar disorder

et al. 2017

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“…Interestingly, patients with RLS with augmentation are at high risk of developing impulsive behavior, suggesting that impulse control disorder and augmentation may share a common pathophysiology [116 && ]. Drug effects: Benzodiazepine may trigger/ exacerbate sleepwalking, occasionally also NSRED; antidepressants may exacerbate RBD, RLS, sleepwalking, and bruxism; neuroleptics can exacerbate RLS and bruxism; dopaminergic drugs may trigger/aggravate NSRED and bruxism [117,118].…”

Section: Etiologymentioning

confidence: 99%

Sleep-related movement disorders and disturbances of motor control

Bargiotas

Bassetti²

2017

Current Opinion in Neurology

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Purpose of reviewReview of the literature pertaining to clinical presentation, classification, epidemiology, pathophysiology, diagnosis, and treatment of sleep-related movement disorders and disturbances of motor control. Recent findingsSleep-related movement disorders and disturbances of motor control are typically characterized by positive motor symptoms and are often associated with sleep disturbances and consequent daytime symptoms (e.g. fatigue, sleepiness). They often represent the first or main manifestation of underlying disorders of the central nervous system, which require specific work-up and treatment. Diverse and often combined cause factors have been identified. Although recent data provide some evidence regarding abnormal activation and/or disinhibition of motor circuits during sleep, for the majority of these disorders the pathogenetic mechanisms remain speculative. The differential diagnosis is sometimes difficult and misdiagnoses are not infrequent. The diagnosis is based on clinical and video-polysomnographic findings. Treatment of sleeprelated motor disturbances with few exceptions (e.g. restless legs/limbs syndrome) are based mainly on anecdotal reports or small series. SummaryMore state-of-the-art studies on the cause, pathophysiology, and treatment of sleep-related movement disorders and disturbances of motor control are needed.

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“…3,4 It has been proposed that bruxism aetiology may be multifactorial and that several underlying mechanisms might play a role in its genesis, such as psychosocial (eg stress and anxiety), physiological (eg genetics) and exogenous factors (eg alcohol consumption, medication use, smoking). 5,6 More importantly, although existing knowledge is still limited, associated factors are thought to be distinct regarding both circadian manifestations of bruxism. Whilst psychosocial aspects appear to have some influence on AB, 7 autonomic/ central nervous system activation might be the primary factors involved in SB genesis.…”

Section: Introductionmentioning

confidence: 99%

Bruxism: An umbrella review of systematic reviews

Melo

Duarte

Pauletto

et al. 2019

J of Oral Rehabilitation

162

136

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Objectives To synthesise available knowledge about both sleep (SB) and awake bruxism (AB) as depicted by previous published systematic reviews (SR). Methods SR investigating any bruxism‐related outcome were selected in a two‐phase process. Searches were performed on seven main electronic databases and a partial grey literature search on three databases. Risk of bias of included SR was assessed using the “University of Bristol's tool for assessing risk of bias in SR”. Results From 1038 studies, 41 SR were included. Findings from these SR suggested that (a) among adults, prevalence of AB was 22%‐30%, SB (1%‐15%), and SB among children and adolescents (3%‐49%); (b) factors consistently associated with bruxism were use of alcohol, caffeine, tobacco, some psychotropic medications, oesophageal acidification and second‐hand smoke; temporomandibular disorder signs and symptoms presented a plausible association; (c) portable diagnostic devices showed overall higher values of specificity (0.83‐1.00) and sensitivity (0.40‐1.00); (d) bruxism might result in biomechanical complications regarding dental implants; however, evidence was inconclusive regarding other dental restorations and periodontal impact; (e) occlusal appliances were considered effective for bruxism management, although current evidence was considered weak regarding other therapies. Conclusions Current knowledge from SR was mostly related to SB. Higher prevalence rates were found in children and adolescents than in adults. Associated factors and bruxism effects on stomatognathic structures were considerably heterogeneous and inconsistent. Overall good accuracy regarding portable diagnostic devices was found. Interventions' effectiveness was mostly inconclusive regarding the majority of available therapies, with the exception of occlusal appliances.

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Psychotropic drugs and bruxism

Cited by 72 publications

References 50 publications

Signs of bruxism and temporomandibular disorders among patients with bipolar disorder

Signs of bruxism and temporomandibular disorders among patients with bipolar disorder

Sleep-related movement disorders and disturbances of motor control

Bruxism: An umbrella review of systematic reviews

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