1985
DOI: 10.1016/0002-9394(85)90685-3
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Secondary Angle-Closure Glaucoma After Central Retinal Vein Occlusion

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Cited by 16 publications
(4 citation statements)
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“…This finding supports the notion that PAC/PACG induces RVO, rather than the reverse association, although it has been proposed that in some cases of CRVO, vascular engorgement and oedema of the posterior segment results in anterior rotation of the cilio‐lenticular diaphragm, thereby closing a previously narrow but open drainage angle (Grant ; Mendelsohn et al. ; Wu et al. ).…”
Section: Discussionsupporting
confidence: 82%
“…This finding supports the notion that PAC/PACG induces RVO, rather than the reverse association, although it has been proposed that in some cases of CRVO, vascular engorgement and oedema of the posterior segment results in anterior rotation of the cilio‐lenticular diaphragm, thereby closing a previously narrow but open drainage angle (Grant ; Mendelsohn et al. ; Wu et al. ).…”
Section: Discussionsupporting
confidence: 82%
“…This form of glaucoma must be distinguished from NVG because the treatments and prognoses may differ between the 2 conditions. General Characteristics [24][25] 1) unilateral acute OH with angle-closure and flattening of the anterior chamber, which appears within 1mo of a CRVO attack; 2) the congener eye shows a deep anterior chamber, normal IOP, and a wide open (45º width) anterior chamber angle; 3) transient character; and 4) lack of the intraocular NV. The shallowness of the anterior chamber is due to an abnormal accumulation of blood or transudative fluid from the retinal vessels, in the posterior segment of the eye, that pushes the vitreous and lens forward, blocks the pupil, and leads to angle-closure glaucoma.…”
Section: Transient Secondary Non-rubeotic Angle-closure Glaucoma Foll...mentioning
confidence: 99%
“…Anti-VEGF agents are beneficial because they act synergistically reversing the increased vascular permeability mediated by VEGF and decreasing the amount of fluid in the posterior portion of the eyeball. Choroidal hemorrhages, massive vitreous hemorrhages, uveal effusions, and PRPs are other causes of transient unilateral flattening of the anterior chamber, with or without angleclosure glaucoma [25] . The reported incidence of non-rubeotic angle-closure glaucoma in patients with CRVO is extremely low (0.68%) [24] .…”
Section: Transient Secondary Non-rubeotic Angle-closure Glaucoma Foll...mentioning
confidence: 99%
“…It has been proposed that in some cases of CRVO, vascular engorgement and edema of the posterior segment may result in anterior rotation of the cilio-lenticular diaphragm, thereby closing a previously narrow but open drainage angle. 11,12 However, it is also plausible that the profound pressure rises that occur in primary angle-closure (PAC) induce CRVO through a similar mechanism to that suggested in POAG. [1][2][3] Furthermore, since the first reports of angle-closure and RVO, 7,8 there have been limited additional data, with a more recent case series of 4 subjects with vascular accidents associated with angle-closure describing only 2 cases of APAC and CRVO.…”
mentioning
confidence: 96%