Sudden decrease in vision caused by liquefied after-cataract

Ghanem, Vinı́cius Coral; Ghanem, Emir Amin

doi:10.1016/s0886-3350(02)01358-5

Cited by 8 publications

(6 citation statements)

References 4 publications

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“…5,12,13 Patients with late postoperative CBS have decreased visual acuity, impaired contrast sensitivity, and glare disability because of PCO and accumulated fluid in the bag. [3][4][5][6]10,[12][13][14] In our cases, no patient reported these symptoms except gradually decreased vision. We think the milky-white fluid may have a more negative effect on contrast sensitivity than visual acuity, such as early nuclear cataract.…”

Section: Discussionmentioning

confidence: 48%

“…If the IOL is placed in the sulcus, entrapment of fluid can be seen between the anterior capsule and the posterior surface of the IOL optic. [2][3][4] Miyake et al 1,2 classified CBS as intraoperative, early postoperative, and late postoperative. Also, they report the main cause of decreased vision after cataract surgery is aftercataract and describe 4 types of aftercataract; that is, fibrous, Elschnig pearl, Soemmerring ring, and liquefied.…”

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confidence: 99%

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Late capsular block syndrome presenting with posterior capsule opacification

Pınarcı

Bayar

Canan

et al. 2012

Journal of Cataract and Refractive Surgery

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Section: Discussionmentioning

confidence: 48%

mentioning

confidence: 99%

Late capsular block syndrome presenting with posterior capsule opacification

Pınarcı

Bayar

Canan

et al. 2012

Journal of Cataract and Refractive Surgery

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“…[156] Onset of vision loss in LAC is gradual, but acute onset vision loss was also reported. [78] Vision loss may be due to scattering of light by the opalescent meniscus posterior to the IOL exerting a piggy bag IOL effect.…”

Section: Discussionmentioning

confidence: 99%

Liquefied after cataract and its surgical treatment

Bhattacharjee

et al. 2014

Indian J Ophthalmol

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Aims:To describe liquefied after cataract (LAC) and its surgical management following an uneventful phacoemulsification with posterior chamber in-the-bag intraocular lens (IOL) implantation and continuous curvilinear capsulorrhexis (CCC).Design:Interventional case series.Materials and Methods:Eleven patients with LAC, following uneventful phacoemulsification with CCC and in-the-bag IOL implantation were enrolled. After the basic slit lamp examination, each case was investigated with Scheimpflug photography and ultrasound biomicroscopy (UBM). Each case was treated with capsular lavage. Biochemical composition of the milky fluid was evaluated and ring of anterior capsular opacity (ACO) was examined under electron microscope.Results:All 11 cases presented with blurring of vision after 6-8 years of cataract surgery with IOL implantation. All cases had IOL microvacuoles, 360° anterior capsule, and anterior IOL surface touch along with ACO, ring of Soemmering, and posterior capsule distension filled with opalescent milky fluid with whitish floppy or crystalline deposits. Biochemically, the milky fluid contained protein (800 mg/dl), albumin (100 mg/dl), sugar (105 mg/dl), and calcium (0.13%) and was bacteriologically sterile. Histologically, the dissected ACO showed fibrous tissue. All cases were successfully treated with capsular lavage with good visual recovery and with no complication. There was no recurrence of LAC during 2 years postoperative follow-up in any of the cases.Conclusions:LAC is a late complication of standard cataract surgery. It may be a spectrum of capsular bag distension syndrome (CBDS) without shallow anterior chamber and secondary glaucoma. Capsular bag lavage is a simple and effective treatment for LAC and a safe alternative to neodymium-doped yttrium aluminum garnet (Nd-YAG) capsulotomy.

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“…1 Other authors report entrapment of liquefied after-cataract between the posterior surface of a sulcus-placed optic and the anterior capsule. 2 When the fluid amount is small, the liquefied aftercataract may be localized between the IOL and the posterior capsule. 3 The fluid contains collagens produced by residual lens epithelial cells (LECs) or necrotic and apoptotic autolyzed LECs.…”

Section: Discussionmentioning

confidence: 99%

Late postoperative capsular block syndrome: Entrapment of liquefied after-cataract by capsular bend

Wang

Cruz²

2005

Journal of Cataract and Refractive Surgery

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We report a case of capsular bend-related entrapment of liquefied after-cataract that resulted in late postoperative capsular block syndrome in a 56-year-old man. Slitlamp examination showed a capsular bend formation at the square edge of the optic. Superiorly, leakage in the capsular bend resulted in fluid extending into Soemmering's ring, although communication with the anterior chamber was limited by the capsular bend. Fluid was seen between the lens and posterior capsule.

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Sudden decrease in vision caused by liquefied after-cataract

Cited by 8 publications

References 4 publications

Late capsular block syndrome presenting with posterior capsule opacification

Late capsular block syndrome presenting with posterior capsule opacification

Liquefied after cataract and its surgical treatment

Late postoperative capsular block syndrome: Entrapment of liquefied after-cataract by capsular bend

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