Symptomatic accessory lobe of liver with a review of the literature

Pujari, B. D.; Deodhare, S. G.

doi:10.1136/pgmj.52.606.234

Cited by 54 publications

(33 citation statements)

References 8 publications

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“…Presence of a large symptomatic accessory liver lobe was found in a woman aged 70 years by Llorente & Dardik (1971). Pujari & Deodhare (1976) have reported the presence of a symptomatic accessory lobe. The accessory lobes may herniate into the thorax through the diaphragm and cause serious problems (Feist & Lasser, 1959), Joshi et al (2009) did an extensive study on variations of the liver.…”

Section: Discussionmentioning

confidence: 90%

A Study on the Anomalies of Liver in the South Indian Cadavers

Nayak

2013

Int. J. Morphol.

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Estudio sobre las Anomalías del Hígado en Cadáveres del Sur de IndiaSatheesha Nayak B. NAYAK, B. S.A study on the anomalies of Liver in the South Indian cadavers. Int. J. Morphol., 31(2):658-661, 2013. SUMMARY:Liver is the largest gland in the body and rarely shows variations in its lobes and fissures. We studied 55 Formalinfixed livers for occurrence of abnormal shape, lobes, fissures and position of gall bladder. In 60% of cases the liver was normal. 40% livers showed one or the other variations. Additional lobes were found in 9.09% of cases. Additional fissure was found in 1.81% of cases. Presence of dumbbell shaped caudate lobe and presence of large papillary process was noted in 1.81% cases each. In 1.81% cases the fissure for ligamentum teres was absent or situated on the anterior surface of the liver. In 1.81% of cases each the liver was either extremely long or extremely flat. In 18.18% of the cases, the gall bladder was short and its fundus did not project beyond the inferior border of the liver. These findings may be useful for surgeons and radiologists.

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Section: Discussionmentioning

confidence: 90%

A Study on the Anomalies of Liver in the South Indian Cadavers

Nayak

2013

Int. J. Morphol.

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“…As a type of congenital anatomical malformation, ALL occurs very rarely because it is associated with an autosomal recessive gene with a very low frequency. Anatomical research through necropsies of 172 rats confirmed this genetic theory (8). Currently, there are two hypotheses of the mechanism of an ALL: (i) the embryonic liver curls outwards and forms an accessory lobe during the embryonic stage of development (10) or (ii) an accessory lobe arises from intra-abdominal hypertension caused by the development of the tunica muscularis recti and the enlargement of the liver (11).…”

Section: The Mechanism Of Allmentioning

confidence: 97%

“…During the 7th and the 8th week of embryo development, which is when the muscular layer of the abdominal wall is formed, development of an ALL in the embryo may obstruct the closing of the umbilical ring (3,10), which is why most ALLs are associated with acromphalus. ALLs are also associated with congenital biliary atresia (22), congenital diaphragmatic defects, and angiocavernoma (8). Most reported cases of an ALL involve females ranging in age from newborns to 75 years.…”

Section: Clinical Featuresmentioning

confidence: 99%

Accessory lobes of the liver: A report of 3 cases and review of the literature

Wang

Cheng

Zhang

et al. 2012

Intractable Rare Dis Res

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“…2 Ectopic liver tissue occasionally causes unexpected clinical problems. [6][7][8] We encountered two patients with an ectopic liver; in one patient, ectopic liver tissue in the stomach developed hepatocellular carcinoma (HCC) while the mother liver had no HCC, and in the other patient with cirrhosis, who died in an automobile accident, an ectopic liver attached to the gallbladder had cirrhosis histologically. A review of the literature revealed more than 20 cases of HCC arising outside the liver, mainly from Japan.…”

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confidence: 99%

Propensity of Ectopic Liver to Hepatocarcinogenesis: Case Reports and A Review of the Literature

Arakawa

Kimura²,

Sakata³

et al. 1999

Hepatology

149

178

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Two patients with ectopic liver are described. In one patient, a small ectopic liver attached to the gastric serosa developed hepatocellular carcinoma (HCC). The preoperative diagnosis was an ␣-fetoprotein (AFP)-producing carcinoma and a malignant ulcer of the stomach. Total gastrectomy and esophago-jejunostomy were performed. The tumor that measured 4 ؋ 2 ؋ 2 cm contained an AFP-producing HCC and normal liver tissue. In another patient who had alcoholic cirrhosis, ectopic liver on the serosa of the gallbladder was found to have the same histological changes as the mother liver. A survey of the literature disclosed more than 20 cases in which HCC developed outside the liver; the liver did not have HCC. By contrast, there was only one report on HCC occurring in the liver in the presence of a noncancerous, relatively large accessory liver lobe. Because ectopic liver does not have a complete vascular and ductal system as a normal liver, it is perhaps functionally handicapped and more prone to hepatocarcinogenesis. (HEPATOLOGY 1999;29:57-61.)

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Symptomatic accessory lobe of liver with a review of the literature

Cited by 54 publications

References 8 publications

A Study on the Anomalies of Liver in the South Indian Cadavers

A Study on the Anomalies of Liver in the South Indian Cadavers

Accessory lobes of the liver: A report of 3 cases and review of the literature

Propensity of Ectopic Liver to Hepatocarcinogenesis: Case Reports and A Review of the Literature

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