The nutritional status of patients with an alcohol problem

Nutritional status in alcoholism

“…Five subjects (16%) were obese as defined by body-weight greater than 120% of reference values. These results are comparable to those of two studies of alcoholic subjects in Scotland (Boyd et al, 1981;Mills et al, 1983), but these Australian subjects appeared to have more normal body-weights than Americans with alcoholic hepatitis (Mendenhall et a]., 1984). In advanced liver disease, it is not possible to separate the direct consequences of malnutrition from the indirect effects of liver disease on nutrition (Mendenhall et al, 1984).…”

The importance of nutrition in aiding recovery from substance use disorders: A review

“…As a result, nutrient deficiencies are prevalent in this population (Chopra and Tiwari, 2012;Stroehle et al, 2012). Research has shown that subjects with AUD are deficient in or have inadequate intake of most nutrients, including: thiamine (Dastur et al, 1975;de la Monte and Kril, 2014;Boyd et al, 1981;Stroehle et al, 6 2012), riboflavin, niacin (Chopra and Tiwari, 2012;Dastur et al, 1975), B 5 (Nabipour et al, 2014), pyridoxine (Dastur et al, 1975;de la Monte and Kril, 2014;Stroehle et al, 2012), folic acid (de la Monte and Kril, 2014;Stroehle et al, 2012;Wu et al, 1975), vitamin A (Clugston et al, 2015;Ross et al, 2012), vitamin C (Boyd et al, 1981), vitamin D (Boyd et al, 1981;Quintero-Platt et al, 2015;Santolaria et al, 2000a;Wijnia et al, 2013;Wilkens Knudsen et al, 2014) vitamin E (Chopra and Tiwari, 2012;Tanner et al, 1986), vitamin K (Iber et al, 1986), magnesium (Dingwall et al, 2015;McLean and Manchip, 1999;Wilkens Knudsen et al, 2014) selenium (Tanner et al, 1986) and zinc, (de la Monte and Kril, 2014;Stroehle et al, 2012;Wilkens Knudsen et al, 2014) (Table 1). Vitamin B 12 may also be deficient; however, circulating levels may not accurately reflect the stores available for use (Kanazawa and Herbert, 1985).…”

Are Diabetic Neuropathy, Retinopathy and Nephropathy Caused by Hyperglycemic Exclusion of Dehydroascorbate Uptake by Glucose Transporters?

“…The incidence of microangiopathy, retinopathy and nephropathy in diabetic patients is directly correlated with low serum ascorbate (Ng et al, 1998;Sinclair et al, 1991;Seghieri et al, 1994;Hirsch et al, 1998;Ali & Chakraborty, 1989;Jennings et al, 1987). Alcoholic patients characterized by low serum ascorbate are also unusually prone to retinopathies, peripheral neuropathies, and nephropathies (Boyd et al, 1981). And finally, scurvy patients do display these pathologies in addition to the bleeding gums, connective tissue disorders and the other symptoms that are more commonly used to characterize the disease (Hurlimann & Salomon, 1994;Hood, 1969 Fig.…”