The Risk of Chronic Kidney Disease Associated with Urolithiasis and its Urological Treatments: A Review

Gambaro, Giovanni; Croppi, Emanuele; Bushinsky, David A.; Jaeger, Philippe; Cupisti, Adamasco; Ticinesi, Andrea; Mazzaferro, Sandro; D’Addessi, Alessandro; Ferraro, Pietro Manuel

doi:10.1016/j.juro.2016.12.135

Cited by 103 publications

(55 citation statements)

References 46 publications

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“…In general, SCFA-producing taxa are responsible for modulation of inflammation and protection against oxidative stress 39. The depletion of Faecalibacterium could thus be involved in the nephrolithiasis-associated chronic kidney injury, which has been recently demonstrated in epidemiological and clinical studies 40. Furthermore, depletion of SCFA-producing taxa has been recently demonstrated in the gut microbiota of patients with chronic kidney disease 41.…”

Section: Discussionmentioning

confidence: 99%

Understanding the gut–kidney axis in nephrolithiasis: an analysis of the gut microbiota composition and functionality of stone formers

Ticinesi¹,

Milani

Guerra³

et al. 2018

Gut

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139

152

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Section: Discussionmentioning

confidence: 99%

Understanding the gut–kidney axis in nephrolithiasis: an analysis of the gut microbiota composition and functionality of stone formers

Ticinesi¹,

Milani

Guerra³

et al. 2018

Gut

Self Cite

139

152

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“…Other risk factors associated with CKD identified in KSF include: existing kidney functional impairment, recurrent urinary tract infections with kidney stones of particular compositions including struvite, UA stones, symptomatic stones, solitary kidney, neurogenic bladder, renal or urinary tract malformations and ileal diversion, malabsorptive bowel conditions and some monogenic diseases responsible for nephrolithiasis and/or nephrocalcinosis, especially in children (Table 1) [73].…”

Section: Kidney Stones As a Multi-systemic Illnessmentioning

confidence: 99%

Meeting report of the “Symposium on kidney stones and mineral metabolism: calcium kidney stones in 2017”

et al. 2019

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A symposium on kidney stones and mineral metabolism held on December 2017 in Brussels, Belgium was the first international multidisciplinary conference of the International Collaborative Network on Kidney Stones and Mineral Metabolism. This meeting addressed epidemiology, underlying pathophysiological mechanisms, genetics, pathological, as well as clinical and research topics. The participants included clinicians and recognized experts in the field from Europe and the United States interacted closely during the symposium which promoted a chance to explore new frontiers in the field of kidney stone disease. This manuscript summarizes some of the major highlights of the meeting.

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“…Risk factors for chronic kidney disease (CKD) include race, gender, age, and family history. Moreover, smoking, obesity, hypertension, diabetes mellitus, systemic inflammatory state/inflammation, and urolithiasis can also lead to CKD [ 3 , 4 , 5 ]. The development of CKD is associated with significant changes in the acid excretion through the kidneys.…”

Section: Introductionmentioning

confidence: 99%

Dietary Treatment of Metabolic Acidosis in Chronic Kidney Disease

Siener¹

2018

Nutrients

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Chronic kidney disease and reduced glomerular filtration rate are risk factors for the development of chronic metabolic acidosis. The prevention or correction of chronic metabolic acidosis has been found to slow progression of chronic kidney disease. Dietary composition can strongly affect acid–base balance. Major determinants of net endogenous acid production are the generation of large amounts of hydrogen ions, mostly by animal-derived protein, which is counterbalanced by the metabolism of base-producing foods like fruits and vegetables. Alkali therapy of chronic metabolic acidosis can be achieved by providing an alkali-rich diet or oral administration of alkali salts. The primary goal of dietary treatment should be to increase the proportion of fruits and vegetables and to reduce the daily protein intake to 0.8–1.0 g per kg body weight. Diet modifications should begin early, i.e., even in patients with moderate kidney impairment, because usual dietary habits of many developed societies contribute an increased proportion of acid equivalents due to the high intake of protein from animal sources.

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The Risk of Chronic Kidney Disease Associated with Urolithiasis and its Urological Treatments: A Review

Cited by 103 publications

References 46 publications

Understanding the gut–kidney axis in nephrolithiasis: an analysis of the gut microbiota composition and functionality of stone formers

Understanding the gut–kidney axis in nephrolithiasis: an analysis of the gut microbiota composition and functionality of stone formers

Meeting report of the “Symposium on kidney stones and mineral metabolism: calcium kidney stones in 2017”

Dietary Treatment of Metabolic Acidosis in Chronic Kidney Disease

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