“…It allows them to regain advantage over the host by creating new viral strains with acquired resistance to most of the available antiviral medicines. AgNPs of a size close to the size of the virus (20 nm) have an antiviral effect, by penetrating its nucleic acid and destroying or modifying its structure, which prevents a further self-replication [ 97 , 111 , 112 , 113 , 114 , 115 , 116 , 117 , 118 , 119 , 120 , 121 , 122 , 123 , 124 , 125 , 126 , 127 , 128 , 129 , 130 , 131 , 132 , 133 , 134 , 135 , 136 , 137 , 138 , 139 , 140 , 141 , 142 , 143 , 144 , 145 , 146 , 147 , 148 , 149 , 150 , 151 , 152 ]. The course of many viral infections is regulated by very complex interactions between the virus particles and a specific protein synthesis system as a defense of host cells.…”