Vasoactive Catecholamines

Simmons, Jeffery P.; Wohl, James S.

doi:10.1016/b978-1-4160-2591-7.10176-6

Cited by 8 publications

(7 citation statements)

References 0 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…As before, consider the possibility of ongoing fluid loss, hypoxia, electrolyte disorders (hypocalcaemia), hypoglycaemia, acid-base disturbances, hypothermia, cardiac dysfunction (cardiomyopathy and/or arrhythmias), pleural space or pericardial disease, sepsis or SIRS, and institute management as appropriate. 41 Careful attention to reassessment of circulating volume, pain control, oxygen supplementation and cardiac function remains important.…”

Section: Special Considerationsmentioning

confidence: 99%

The Flat Cat

Murphy¹,

Hibbert

2013

Journal of Feline Medicine and Surgery

View full text Add to dashboard Cite

Aim: Detailed information regarding the causes and treatment of acute collapse in the cat can be difficult to locate in a single published source. This two-part review aims to provide a logical approach to the clinical assessment and stabilisation of the critically ill collapsed cat. Practical relevance: Cats are particularly challenging when presented as emergency patients, often in the later stages of an illness or with a vague history and non-specific signs. The nuances of the critically ill cat are considered, especially for shock and its management. Clinical challenges: Shocked cats do not present in the classic stages typically seen in dogs, and the signs are more subtle. Therefore, the clinician must consider whether physical parameters are appropriate for the state and environment of the patient; for example, a normal heart rate in a shocked cat would be considered inappropriate. Audience: This review is directed at any veterinarian working with feline patients, and particularly those dealing with emergencies on a regular basis. Evidence base: There is an extensive body of published literature, both original studies and textbook chapters, pertaining to the causes and treatment of collapse in the cat. In this article the authors draw on information from original publications, reviews and their clinical experience to provide practical guidance to assist in the emergency setting.

show abstract

Section: Special Considerationsmentioning

confidence: 99%

The Flat Cat

Murphy¹,

Hibbert

2013

Journal of Feline Medicine and Surgery

View full text Add to dashboard Cite

show abstract

“…Dobutamine was reported to increase cardiac index and stroke volume while decreasing pulmonary vascular resistance in 10 human patients with VAE‐induced hemodynamic dysfunction 26 . However, dobutamine must be used with caution in cats at doses exceeding 5 μg/kg/min as it has been associated with seizure development 27 …”

Section: Discussionmentioning

confidence: 99%

Full recovery after cardiac arrest secondary to accidental iatrogenic venous air embolism in a cat

Pacifico¹,

Weishaar²,

Boozer³

et al. 2010

Journal of Veterinary Emergency and Critical Care

View full text Add to dashboard Cite

show abstract

“…[4][5][6] The main causes of hypotension include decreased preload, heart disease, and decreased vascular tone, alone or in combination. 7,8 Decreased vascular tone may result from electrolyte abnormalities, hypoxia, acidosis, drugs, toxins, hypothermia, and systemic inflammatory response syndrome attributable to any cause, including sepsis. [5][6][7][8] Silverstein et al 8 reported that critically ill cats that had hypotension (defined as Doppler SAP ≤ 90 mm Hg for purposes of that study) at some time during their hospitalization had a higher mortality rate (25/39 [64%]) than did cats that were normotensive throughout hospitalization (14/44 [32%]).…”

mentioning

confidence: 99%

“…7,8 Decreased vascular tone may result from electrolyte abnormalities, hypoxia, acidosis, drugs, toxins, hypothermia, and systemic inflammatory response syndrome attributable to any cause, including sepsis. [5][6][7][8] Silverstein et al 8 reported that critically ill cats that had hypotension (defined as Doppler SAP ≤ 90 mm Hg for purposes of that study) at some time during their hospitalization had a higher mortality rate (25/39 [64%]) than did cats that were normotensive throughout hospitalization (14/44 [32%]). In addition, hypotensive cats in which the SAP was increased by ≥ 20 mm Hg above the lowest determined value were more likely to survive to discharge than were cats in which the SAP was increased by < 20 mm Hg.…”

mentioning

confidence: 99%