As children develop, they differ from adults in a number of important ways, including anatomy, metabolism, immune system, and the extent of myelination of the nervous system. As a consequence, equivalent exposures to radiation from mobile phones result in different doses to specific tissues in children compared with adults. Higher doses are likely to have more severe implications in the young. A young child's skull is not only smaller and thinner than an adult's, but also has dielectric characteristics closer to those of soft tissues, probably due to a higher water content. The young skull better matches the electromagnetic characteristics of the skin and brain. As a result, finite-difference time-domain (FDTD) simulations confirm field penetration and higher specific absorption rate (SAR) in deeper structures in the young brain. If the peak spatial SAR (psSAR) is modeled in the entire head, as current testing standards recommend, the results for adults and children are equivalent. Our anatomically based evaluations rely on FDTD simulations of different tissues within the brain and confirm that the psSAR in a child's brain is higher than in an adult's brain.
The design, simulations, and optimized results for a novel low specific absorption rate (SAR) monopole antenna on a single artificial magnetic conductor (AMC) cell are described in this paper. Simulated results show a reduction close to 70% in the 1 g ps SAR for the developed monopole antenna with the AMC in comparison to the monopole antenna without AMC. This allows higher radiation efficiency, battery drain reduction as well as mobile terminal user health risks reduction.
In the 1990s, the Institute of Electrical and Electronics Engineers (IEEE) restricted its risk assessment for human exposure to radiofrequency radiation (RFR) in seven ways: (1) Inappropriate focus on heat, ignoring sub-thermal effects. (2) Reliance on exposure experiments performed over very short times. (3) Overlooking time/amplitude characteristics of RFR signals. (4) Ignoring carcinogenicity, hypersensitivity, and other health conditions connected with RFR. (5) Measuring cellphone Specific Absorption Rates (SAR) at arbitrary distances from the head. (6) Averaging SAR doses at volumetric/mass scales irrelevant to health. (7) Using unrealistic simulations for cell phone SAR estimations. Low-cost software and hardware modifications are proposed here for cellular phone RFR exposure mitigation: (1) inhibiting RFR emissions in contact with the body, (2) use of antenna patterns reducing the Percent of Power absorbed in the Head (PPHead) and body and increasing the Percent of Power Radiated for communications (PPR), and (3) automated protocol-based reductions of the number of RFR emissions, their duration, or integrated dose. These inexpensive measures do not fundamentally alter cell phone functions or communications quality. A health threat is scientifically documented at many levels and acknowledged by industries. Yet mitigation of RFR exposures to users does not appear as a priority with most cell phone manufacturers.
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