SUMMARYA case of a 3-month-old male infant with myocardial infarction due to left coronary ostial stenosis is presented.Clinically it was quite similar to anomalous origin of the left coronary artery.Aortogram demonstrated hypoplastic left coronary artery.Postmortem examination revealed quite narrow left coronary ostium and myocardial infarction with aneurysm of the left ventricular apex. AdditionalIndexing Words: Myocardial infarction Coronary ostial stenosis M YOCARDIAL infarction in infancy is rare and may result from one of the several conditions. Aberrant left coronary artery from the pulmonary artery may be the most common cause of myocardial infarction in infancy.1) Recently in Japan acute febrile mucocutaneous lymph node syndrome2),3) is attracted attention as a cause of myocardial infarction in infancy. This report presents an infant who suffered from myocardial infarction due to left coronary ostial stenosis which was clinically similar to anomalous origin of the left coronary artery from the pulmonary artery. CASE REPORTA 10-day-old Japanese male infant was admitted to Nihon University Hospital because of convulsion. He was delivered from a 22-year-old mother at 39 weeks' gestation by Cesarian section because of breech presentation. His birth weight was 2,550Gm. He had been well until 6 days old when he developed convulsion and was admitted to the hospital. Serum calcium level was 7.2mg/100ml, phosphate From the

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A 4-month-old male infant with acute febrile mucocutaneous lymph-node syndrome developed mitral regurgitation, pericardial effusion and multiple pulsating masses in the upper and lower extremities. Two-dimensional echocardiography demonstrated aneurysms in both coronary arteries. Arteriography of the right arm demonstrated multiple aneurysms. Seventeen months later, arteriography and two-dimensional echocardiography demonstrated regression of the aneurysms. This case provides evidence that regression of aneurysms can occur in mucocutaneous lymph node syndrome.

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Five cases of postmyocarditic change with clinical profiles of cardiomyopathy in children are described. Some cases with a history of acute myocarditis show postmyocarditic hypertrophy, which may be related to certain immunologic processes. Others with no apparent clinical history of acute myocarditis present with a clinical picture of hypertrophic cardiomyopathy and show improvement. Based on long-term observation, these cases are considered to represent postmyocarditic hypertrophy. Long-term follow-up with full laboratory examination, including endomyocardial biopsy is considered necessary in establishing the final diagnosis.