Early findings point to the value of early screening to identify patients at risk of treatable pain, physical, and psychological impairments. Moreover, early multidisciplinary intervention models following traumatic injury show promise for protecting against the onset of posttraumatic psychological disorders.

Downsizing is a process to make a company more efficient and costeffective by eliminating nonvalue-added work processes and people. Unfortunately, it has become a common business practice in today's firms in order to stay competitive and strong. The practice, however, can be disastrous if the company does not treat the human aspect of the process. During and after downsizing the workforce may suffer from what is known as the "survivor syndrome". This article addresses the work overload felt by employees in the aftermath of a downsizing. A firm's effective management of downsizing is intricately related to the possible strategies of alleviating work overload and addressing employees' concerns. This relationship is especially important between the organisation and the employees during and after the change. The success or failure of downsizing relies on the remaining workforce.Downsizing affects all individuals involved as well as the organisation as a whole. It sees no difference between the executive officers, managers, the remaining workforce or the ones who find themselves without a job. Thus, it is imperative for a firm to provide the necessary support and services, and as with all types of relationships both personal and organisational, the line of communication must be open and truthful. Volume 27 Number 4/5 2004 9 New Development Concerning the Effect of Work Overload on Employees * Communicate the new direction of the company to the employees. Show them their abilities to contribute and be a part of Volume 27 Number 4/5 2004 11 New Development Concerning the Effect of Work Overload on Employees

There is evidence that individuals with an acquired brain injury (ABI) are at increased risk of developing psychological problems and that they commonly experience difficulties in social communication, associated with poorer long-term outcomes. Although several relevant group interventions have been evaluated, there has been limited exploration of the feasibility of an ABI inpatient intervention. This nonrandomised pilot study tested the feasibility of an inpatient multidisciplinary social communication and coping skills group intervention within 1-year post traumatic/nontraumatic ABI. Seven participants completed a 4-week group program (3 × 1 hour sessions per week) facilitated by a speech pathologist and clinical psychologist and were assessed pre/post intervention and at 3 months with the La Trobe Communication Questionnaire, Correct Information Unit analysis, Hospital Anxiety and Depression Scale, Mini International Neuropsychiatric Interview, Coping Self-Efficacy scale and World Health Organization Quality of Life assessment. Most participants improved between baseline and 3 months post intervention in terms of greater informativeness and efficiency of connected speech and reduced anxiety and they provided positive feedback about the group program. Despite the challenges and limitations of this pilot study, the findings are encouraging and support both the value and feasibility of developing such a program into routine inpatient rehabilitation services.

Reflection Fluorescence Microscopy system measured an Mtw1p-Nuf2p fluorescence colocalization of 34 5 2.2%. We performed a similar experiment, immobilizing the kinetochores onto microtubules. We measured a colocalization on microtubules of 8156.4% indicating that Nuf2p remained bound to the kinetochore upon kinetochore-microtubule attachment in vitro, as expected. We repeated this experiment for kinetochores with fluorescently labeled Mtw1p and Bub1p, a SAC component. We measured a 12 5 3.4% Mtw1p-Bub1p colocalization when the particles were immobilized onto a coverslip and 50 5 1.9% on microtubules. These experiments indicate that kinetochore-microtubule attachment in yeast may not be sufficient to release Bub1p. Current efforts are focused on Mad1p, a checkpoint protein that is believed to be released from kinetochores upon microtubule attachment in vivo.