Left atrial appendage (LAA) aneurysm is an extremely rare anomaly. So far, less than one hundred cases only have been reported worldwide. Revelation modes are dominated by complications such as arrhythmias and thromboembolic events. We herein report a pediatric case of huge congenital LAA aneurysm with an original revelation mode that has never been described before in medical literature.
Paradoxical embolism in coronary artery is a rarely diagnosed clinical entity. In the majority of reported cases; the diagnostic of this pathology is « presumptive » based on certain criteria. It can be considered “proven” when the embolus is found lodged in the abnormal communication between the venous and arterial circulation; which is very rare. We herein report a case of myocardial infarction caused by a proven paradoxical coronary embolism through a patent foramen ovale. The authors highlight through this paper the contribution of echocardiography and particularly trans-esophageal echocardiography, especially if performed soon after presentation, for early diagnosis.
Background: Behcets disease is a systemic vasculitis with unusual thrombosis, especially in children. Intracardiac localization is rare. The site of choice is the right ventricle. It may be isolated, but must necessarily seek other vascular attacks, in particular an aneurysmal pathology of pulmonary arteries. Its clinical expression is not specific.
Case Presentation: An 8-years-old boy was hospitalized for an isolated prolonged fever with marked inflammatory state. No infectious symptoms were identified. Electrocardiogram showed a right branch block and negative T waves in V1-V3 leads. Transthoracic echocardiography revealed multiple right ventricular masses, associated with minimal pericardial effusion. These masses have various sizes and are lining right side of interventricular septum and pulmonary infundibulum without right ventricular outflow tract obstruction. Thoracic computerized tomography scan and cardiac magnetic resonance imaging confirmed that cardiac masses were multiples thrombi filling right ventricle and pulmonary artery. Thrombophilia panel assessment and eye fundus examination were normal. The patient was not a carrier of the HLA B51 gene. Juvenile Behcets disease was the final diagnostic. Treated by anticoagulant and corticotherapy, the clinical and ultrasound course of the patient was favorable. A 3-year follow up didnt show a recurrence of these thrombi.
Conclusion: Regression of thrombus under anticoagulant, of fever and inflammatory syndrome under corticosteroid therapy, is a good retrospective diagnostic criterion.
Authors report a case of a malignant psoas tumor diagnosed in a 69-year-old patient with an abdominopelvic CT scan as well as a PET-Scanner performed as part of the assessment of infective endocarditis extensions on aortic stenosis. Authors insist on the rarity of psoas tumors which remain mostly malignant, and on the interest of the CT scan and percutaneous biopsy for a precise histological diagnosis conditioning the therapeutic management. Treatment must be integrated into multidisciplinary approach where surgery occupies preponderant place. This case illustrates a therapeutic quandary regarding to timing of cardiovascular surgery related to carcinological surgery and the complement by radio and chemotherapy.
Residual atrial septal defect (ASD) is a rare complication after percutaneous closure of an ostium secundum atrial defect in eligible patients. Cardiac imaging, mainly transthoracic and transesophageal echocardiography is essential to establish the diagnosis. Surgical closure is the therapeutic strategy of choice. We present the case of a 54-year-old woman who presented with a residual left-to-right atrial shunt four years after percutaneous closure of an ASD ostium secundum.
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