Acinetobacter genus includes multiple species, most notably A. baumanii that constitutes a common cause of nosocomial infections worldwide, particularly in patients with underlying immunodeficiency and risk factors (e.g., prior broad-spectrum antibiotic therapy, central venous catheter, mechanical ventilation). A. junii is a very rare human pathogen that is particularly associated with outbreaks of sepsis in immunocompromised neonates and pediatric oncology patients and rarely in immunocompromised adults. To our knowledge, this is the first case report of cavitary pneumonia with bacteremia secondary to A. junii in a patient with systemic lupus erythematosus (SLE).

Solitary plasmacytoma (SP) is characterized by an accumulation of neoplastic monoclonal plasma cells in a localized fashion, without evidence of multiple myeloma. It makes up <5% of all plasma cell neoplasms and is typically found in regions like the pelvis, ribs, vertebra, and spine. SP is classified into extramedullary plasmacytoma (EMP), which primarily affects soft tissues, and solitary bone plasmacytoma (SBP), which primarily affects the pelvis, ribs, vertebrae, and spine. We report a case of a 66-year-old man with sternal plasmacytoma presenting as chest pain. He was treated with radiation therapy. Here, we aim to describe the clinical features, diagnostic methods, treatment, and potential outcome in a patient with SBP.

Peritumoral light chain (AL) amyloidosis secondary to lymphoid malignancies is a rare but well-described entity. Peritumoral deposition of amyloid without systemic amyloidosis has been described in mucosaassociated lymphoid tissue (MALT) lymphomas; however, there are no reported cases of follicular lymphoma with localized peritumoral AL amyloidosis without systemic involvement of amyloidosis. We present a rare case of a patient with advanced follicular lymphoma with peritumoral lymph node IgM lambda light chain amyloidosis without an underlying monoclonal gammopathy or plasma cell dyscrasia.

with extensive pneumatosis (Figure A), consistent with cecal bascule (type III cecal volvulus). The ascending colon was dilated up to a decompressed hepatic flexure, where nonspecific bowel wall thickening was visualized. She had a normal colonoscopy a few months prior. The patient was initially managed conservatively, however a lower gastrointestinal series showed persistent obstruction at the hepatic flexure with bird-beak appearance (Figure B). She underwent an exploratory laparotomy that discovered a mass in the right upper quadrant involving the gallbladder and liver, causing extrinsic compression of the hepatic flexure and numerous omental nodules. Biopsies confirmed metastatic cholangiocarcinoma. The patient began evaluation for cancer staging, but ultimately passed one month after diagnosis. Discussion: The development of a cecal bascule is usually attributed to an abnormally mobile cecum due to improper development and insufficient fixation of the mesentery to the posterior parietal peritoneum. A mobile cecum may also be acquired in pregnancy due to enlargement of the uterus, or after abdominal surgery such as open appendectomies which require extensive division of peritoneal attachments to the cecum. Clinical presentation can be variable with abdominal pain, distention and nausea being most common symptoms and ranging to an acute abdomen and perforation. Diagnosis of type III cecal volvulus requires CT imaging confirming a distended cecum lying anterior to ascending colon. Treatment generally involves surgical resection of the affected bowel as colonoscopic detorsion may lead to recurrence. This is a unique case of an extrinsic malignant obstruction causing a cecal bascule, highlighting the importance of a comprehensive evaluation for underlying etiologies.[2027] Figure 1. A: Cecal bascule with extensive pneumatosis on CT. B: Lower gastrointestinal series showing obstruction at hepatic flexure with bird-beak appearance.