A simple wideband bandstop filter which yields equal ripples in the stopband is described. The filter comprises only two resonators and a connecting transmission line. Ultra-wide stopband characteristic is achieved by realising three transmission zeros near the passband edges. Stopband rejection depth and bandwidth can be controlled by impedances of the configuration. The defected ground structure is chosen to realise the high impedance line. To validate the proposed topology, a compact microstrip bandstop filter centred at 1.5 GHz with a measured 20 dB rejection bandwidth of about 139% is fabricated.

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Mibefradil inhibition of the bioelectrical signal and uterine contractile forces suggest that T-type calcium channels are important in the initiation of each contraction. Some results using indomethacin suggest that for a specific tissue or under some conditions, autocrine stimulation by prostaglandins may be important for recruitment of myocytes in the electrical phase. Stimulation by prostaglandins is likely important in the paracrine phase. Enhanced inhibition by nifedipine in the paracrine phase suggests the mechanism of action of nifedipine tocolytic effect is block of prostaglandin F2alpha (PGF2alpha) paracrine stimulation. Our initial model proposed at least two mechanisms (electrical and nonelectrical) for the recruitment of myocytes. Our modified model suggests that the electrical phase contains a prostaglandin autocrine mechanism in addition to excitation-contraction coupling, and the mechanism in the nonelectrical phase is prostaglandin paracrine signaling.

These findings suggest that the trigger for the initiation of each contraction is a tissue-level bioelectrical event, and some cells are initially recruited to participate in each contraction by excitation-contraction coupling. After the initial phase of the contraction, cells are recruited by a nonelectrical mechanism.

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