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The efficiency of decontamination of aflatoxin residues in poultry feeds through exposure to sunlight (solar radiation), γ-radiation ( 60 Co), and microwave heating were investigated in artificially contaminated feed samples. Photodegradation of aflatoxin by sunlight has been found to cause a significant (P < 0.05) decrease in both B1 and the total aflatoxins. Moreover, the degrees of aflatoxins were dependent on exposure time. Both aflatoxin B1 and total aflatoxins were decreased when feed samples exposed to sunlight by 42.3, 39.9, 75.5, and 65.9% for 3 and 30 h of direct sunlight of the treatment T1, whereas feed samples subjected to γ-irradiation and microwave heating caused a significant (P < 0.05) decrease in aflatoxin B1 contents by 42.7 and 32.3% for γ-irradiation and microwave heating (T3 of 25 kGy and 10 min of microwave heating), respectively. Therefore, the solar radiation was more effective in aflatoxin B1 reduction when compared with γ-irradiation and microwave heating.

The conventional heating methods of milk did not cause any significant increase in the trans isomer content, with the exception of milk heated at 63 +/- 1.0 degrees C for 30 min and milk microwaved for 5 min, which were significantly increased by 19 and 31%, respectively. The chemical changes of lipids were generally accelerated with the severity of the heat treatment and duration of storage. The conjugated linoleic acid content of cheese heated in a microwave oven for 5 min decreased by 21%, and microwave heating for 10 min caused a decrease of 53% compared with that of freshly boiled cheese.

Aflatoxin B1(AFB1) and total Aflatoxins (AFT) contaminated feed effect on aflatoxins residue level in eggs, muscles (breast, leg), organs (liver, kidney, gizzard) and excreted aflatoxins in chicken litter of layer hens were monitored. Laying hens were on four levels of aflatoxins for 6 weeks and monitored weekly for the change in both AFB1 and AFT levels. Pronouncedly, the AFB1 and AFT were detected in eggs, muscles (legs, breast), organs (liver, kidney and gizzard) and litter in noticeable amounts. Total Aflatoxin (AFT) level was lowest in chicken breast (0.63 ppb) and highest in liver (2.12 ppb) and gizzard (1.22 ppb) of chicken fed diet with 965.12 ppb. Whereas, AFB1 residue was 0.66 ppb in eggs, 1.59 ppb in liver tissues of hens given feed contaminated with 894.12 ppb. Residue level of AFB1 was high in liver and kidney of all treatments. The chicken breast tissues were lowest in AFB1 and AFT of values 0.72 and 0.63, respectively. Eggs production was significantly (p<0.05) affected with AFB1 contaminated feed and egg production was decreased by more than 30%

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