Tumors of the salivary gland constitute a heterogeneous group of variable histological and biological behaviors. Patients with salivary gland tumors typically present with painless swelling. However, several neoplastic and non-neoplastic pathologies can result in salivary gland enlargement. We report the case of a 35-year-old woman complaining of a left neck swelling for 3 months duration. She had no relevant past medical or surgical history. On examination, there was a left submandibular swelling that was firm in consistency, non-tender, non-pulsatile, relatively mobile, and was not tethered to the underlying structures. Otherwise, examination of the head and neck was unremarkable. A CT scan of the neck revealed a welldefined hypodense lesion in the left submandibular region with foci of calcification along with multiple enlarged lymph nodes. After surgical exploration, the submandibular gland region, a mass lesion was found arising from the submandibular gland. Histopathological examination revealed the diagnosis of schwannoma. Salivary gland schwannoma is a very rare form of neurogenic tumor. Surgical resection is the treatment of choice; however, neural deficits are important and common postoperative complications.

Necrotizing sialometaplasia (NS) is defined as a benign, self-limiting inflammatory condition of tissues related to salivary gland. It accounts for less than 1percent of total of biopsied oral lesions. Necrotizing sialometaplasia (NS) has a very good prognosis. Ischemia owing to trauma is the most common cause of necrotizing sialometaplasia, which is a perfectly benign lesion. With or without biopsy, the lesions heal on their own. The goal of a biopsy is to rule out cancer as the cause of their troubling clinical symptoms. The majority of cases of necrotizing sialometaplasia are thought to be caused by vascular ischemia. Management of Necrotizing sialometaplasia doesn’t necessates surgery or medications as the disease is self-healed one, surgery is only applied when cellular masses are massive. This article aims to overview epidemiology, diagnosis and management of necrotizing sialometaplasia.

Total edentulism is a serious public health issue, particularly among the aged people.  Edentulism has a detrimental impact on various elements of a patient's everyday life, including masticatory function, phonetics, and facial structure, with significant psychosocial implications. For those patients, complete dentures remain the first option. Despite the fact that age-specific rates of edentulism are expected to decline, demand for complete dentures will continue to rise in the next decades. Patients with new complete dentures who are edentulous are fairly pleased, however up to 30% of them have issues. They continue to struggle with issues such as unattractive appearance, ripping pain or discomfort owing to the failure of holding and stabilization, impaired articulation, food deposition under the denture, and trouble with chewing. These types of issues can lower one's quality of life and force him to seek additional help to resolve them. In some circumstances, a specific clinical technique known as rebasing is recommended as a solution to these issues.

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Intramural hematoma of the esophagus (IHE) or dissecting intramural hematoma is a relatively unusual complication of acute mucosal and submucosal lesions that results in a blood accumulation between the layers of the esophagus. Esophageal hematoma is a rare condition that can develop spontaneously or as a result of trauma, poisoning, or medical intervention. Mallory-Weiss syndrome, Boerhaave syndrome, and IHE are all forms of acute mucosal damage of the esophagus, with IHE being the rarest of the three. In general, esophageal traumatic damage, including traumatic penetration and perforation, is uncommon, making IHE incidence and prevalence difficult to measure. Although most esophageal hematomas are asymptomatic, they can cause significant chest discomfort, dysphagia, and hematemesis. Esophageal hematomas should be distinguished from Mallory-Weiss tear and Boorhaave's syndrome, which they may closely resemble in such cases. Moreover, cardiovascular and respiratory diseases should be ruled out, therefore further tests such as an electrocardiogram, chest X-ray, and laboratory testing can be useful. The majority of cases resolve with conservative treatments, with symptoms disappearing in 1-2 weeks. NPO, IV fluids, acid suppression, and treatment of coagulopathy are all conservative procedures. This review aims to summarize current evidence on etiology, epidemiology, diagnosis and management of esophageal hematoma.