No abstract

Pronto Dry seems promising as a diagnostic tool to detect H pylori more rapidly and accurately.

Background Helicobacter pylori (HP) can colonise beneath layer of mucous on the surface of gastric epithelial cells caused chronic infection more than 95% at antrum (chronic antral inflamation) besides corpus with spreading inequitably. This inflamation can cause gastric lession as a Gastritis, Gastric Ulcer (GU), Cancer of Duodenal Ulcer (DU). Absence of chronic antral inflamation (increased number of mononuclear cells in lamina propia) is a highly specific methode to exclude H. pylori infection. Objective To investigated endoscopic mucosal lesion (Gastritis, GU, Cancer or DU) and histologic picture of gastric mucosal H. pylori infection. Methods This study was performed in 50 patient with gastroscopic lesion (Gastritis, GU,Cancer or DU) and CLO test positive. H. pylori was evaluated by modified Giemsa staining and gastritis by Haemotoxiline and eosin (H and E) staining in the antrum and corpus. Result Reported in The Third Western Pasific Helicobacter Congress, June 22‐24 Bali Indonesia.

Introduction The prevalence of H. pylori infection in Indonesia (Medan 52, Jakarta 40‐57, Mataram 51‐66%). And Non Serological Diagnostic test for H. pylori, with methods Invasive (CLO, Microbiology and Histopatology) and Non invasive (UBT), but cost this method is expensive for the lower class and rural area. Background H. pylori produces of Urease enzymes and urease activity is an important characteristic of the Organism, so How about Urea and Ammonium concentration in Gastric juice patients H. pylori infection? Objective Gastric juice analyzed for Urea and Ammonium H. pylori Infection, compare with non infection H. pylori, and the future Gastric juice aspiration to analyze by Naso gastric tube ?. Material and methods Patients with chronic dyspepsia, without consumption antibiotics, omeprazole/H2RA within 1 month, no ureami and investigation with upper endoscopi to gastric juice aspiration and CLO test. Gastric juice analyzed by colorimetric method and spectrophotometric assay. Patients control: no symptom dyspepsia, IgG anti HP negative, Upper endoscopy normal. Result Preliminary report H. pylori positive‐patients bad lower urea and higher ammonia concentration compared with non infection H. pylori (control) Conclusion The Third Western Pacific Helicobacter Congress, June 22‐24 Bali‐Indonesia.

Background Helicobacter pylori (HP) can colonise beneath layer of mucous on the surface of gastric epithelial cell caused chronic infection more than 95% at antrum (chronic antral inflammation) besides corpus with spreading inequitably. This inflammation can cause gastric lesion as a Gastritis Gastric Ulcer (GU), Duodenal Ulcer (DU) or cancer. Absence of chronic antral inflammation (increased number of mononuclear cells in lamina propria) is a highly specific methode to exclude HP infection. Objective To investigated endoscopic mucosal lesion in Gastritis and histologic picture of gastric mucosal HP infection Method This study was performed in 50 patient with gastroscopic lesion with Gastritis and CLO test positive HP was evaluated by modified Giemsa staining and gastritis by Hematoxiline and eosin (H and E) staining in the antrum and corpus. Result Reported in the Third Western Pacific Helicobacter Congress, June 22‐24 Bali Indonesia.