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The article contains sections titled: 1. Organic Bromine Compounds 1.1. Physical Properties 1.2. Chemical Properties 1.2.1. Nucleophilic Displacement of Bromine 1.2.2. Displacement of Bromine by Metals 1.3. Production 1.3.1. Addition Reactions 1.3.2. Substitution Reactions 1.4. Commercial Products 1.4.1. 1,2‐Dibromoethane 1.4.2. 1‐Bromododecane 1.4.3. 1‐Bromo‐3‐chloropropane 1.4.4. 1‐Bromo‐2‐phenylethane 1.4.5. 1‐(Bromomethyl)‐3‐phenoxybenzene 1.4.6. Bromoacetic Acid 1.4.7. Bromomethane 1.4.8. 3‐Bromo‐1‐propene 1.4.9. Bromochloromethane and Dibromomethane 1.4.10. Trifluorobromomethane and Difluorobromochloromethane 1.4.11. Flame Retardants 1.4.11.1. Tetrabromobisphenol‐A 1.4.11.2. Decabromobiphenyl Oxide 1.4.11.3. Tetrabromophthalic Anhydride 1.4.11.4. Hexabromocyclododecane 1.5. Manufacturers 1.6. Toxicology and Occupational Health 2. Inorganic Bromine Compounds 2.1. Hydrogen Bromide and Bromides 2.1.1. Hydrogen Bromide 2.1.2. Bromides 2.2. Hypobromous Acid, Hypobromites 2.3. Bromous Acid, Bromites 2.4. Bromic Acid, Bromates

The article contains sections titled: 1. Organic Bromine Compounds 1.1. Physical Properties 1.2. Chemical Properties 1.2.1. Nucleophilic Displacement of Bromine 1.2.2. Displacement of Bromine by Metals 1.3. Production 1.3.1. Addition Reactions 1.3.2. Substitution Reactions 1.4. Commercial Products 1.4.1. 1,2‐Dibromoethane 1.4.2. 1‐Bromo‐3‐chloropropane 1.4.3. 1‐Bromo‐2‐phenylethane 1.4.4. 1‐(Bromomethyl)‐3‐phenoxybenzene 1.4.5. Bromoacetic Acid 1.4.6. Bromomethane 1.4.7. 3‐Bromo‐1‐propene 1.4.8. Bromochloromethane and Dibromomethane 1.4.9. Trifluorobromomethane and Difluorobromochloromethane 1.4.10. Flame Retardants 1.4.11. Biocides 1.4.12. Pharmaceuticals 1.4.13. Dyes and Indicators 1.5. Manufacturers 1.6. Toxicology and Occupational Health 2. Inorganic Bromine Compounds 2.1. Hydrogen Bromide and Bromides 2.1.1. Hydrogen Bromide 2.1.2. Bromides 2.2. Hypobromous Acid, Hypobromites 2.3. Bromous Acid, Bromites 2.4. Bromic Acid, Bromates

Plasma and tissue concentrations of warfarin in the rat were measured as a fianction of time following a 10 mg/kg intravenous dose. The mathematical interpretation of the experimental results suggested that the data could be explained in terms of a two-compartment open model. Following equilibration, which occurred within a few minutes after injection, individual tissue levels and plasma levels of warfarin were found to be always directly proportional.

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