“…However, in reality, the stability of the implant would decrease beyond the yield strain of the bone due to excessive microcrack formation and compression necrosis, which both phenomena trigger bone remodeling [25,30,31]. Thus, high degrees of insertion torque must be questioned since elastic theory predicts that excessive strain not only leads to the decrease of biomechanical stability, but also incites negative biologic responses depending on the implant thread design that influence the compression [17]. Such cell-mediated bone resorption and subsequent bone apposition most often occurring from the pristine bone wall toward the implant surface is responsible for what has under theoretical [32] and experimental [33] basis been coined as implant stability dip, where primary stability obtained through the mismatch between implant macrogeometry and surgical instrumentation dimensions is lost due to the cell-mediated interfacial remodeling to be regained through bone apposition [32,34].…”