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We report 2 patients who complained that the left half of faces appeared distorted. A 58-year-old right-handed woman explained that although the left side of the face was clearly visible, it appeared distorted "like a monster." The left eye looked elongated toward the left ear, while the nose appeared to be bent toward the left cheek, and the mouth toward the chin. She did not have any dyslexia in the left or right visual field. A 53-year-old right-handed man described that the left eyelid of people looked swollen as if they had undergone "failed eyelid surgery," while the nose appeared to be bent downward and the left facial outlines either bulged or writhed. This was the same with pictures of others, but was absent with objects. He did not show hemialexia, color anomia, or optic aphasia. Brain MRIs showed splenial infarctions (figure). The prosopometamorphopsia in the right hemifield was likely caused by the disruption of the pathway from the left occipital area to the right hemisphere. Unilateral prosopometamorphopsia could be a dominant hemisphere-specific disconnection sign in which neurologic abnormalities are observed in the ipsilateral side of the dominant hemisphere.1 Our cases provide additional evidence that the splenium of the corpus callosum interconnects visual cortices and the right hemisphere is dominant for integrating facial information.

[(18)F]FDG PET can visualize increased glucose metabolism in a denervated muscle early as 1 week after injury. Therefore, PET could be adopted as a noninvasive imaging modality for acute nerve injuries. In addition, [(18)F]FDG PET may help to understand the role of the nervous system in the control of peripheral tissues.

High-altitude cerebral edema (HACE) is a rare life-threatening condition observed in individuals who climb high altitudes. This report describes the case of a 38-year-old man who recently climbed a 5000-m-high mountain, showing the following radiologic findings at 3 different anatomical locations: 1) increased T2 signal intensity (SI) without restricted diffusion, with full recovery in the posterior limb of the left internal capsule; 2) increased T2 SI with restricted diffusion, with full recovery in the splenium of the corpus callosum; and 3) increased T2 SI with restricted diffusion and microbleeds, resulting in bilateral encephalomalacia in the globus pallidus. Herein, we report the concurrent typical and atypical radiologic findings of this rare condition caused by vasogenic and cytotoxic edema.